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J Thorac Cardiovasc Surg 2007;134:1382-1383
© 2007 The American Association for Thoracic Surgery

Letter to the Editor

Reply to the Editor

Elizabeth O. Johnson, PhDa, Olga Ananiadou, MDb

a Department of Anatomy, Histology and Embryology, University of Ioannina, School of Medicine, Ioannina, Greece
b Department of Cardiovascular Surgery, University of Ioannina, School of Medicine, Ioannina, Greece

The first 20% of the full text of this article appears below.

Although the deleterious effects of prolonged exposure to cardiopulmonary bypass on brain function and structure have been demonstrated, neuroprotective strategies, particularly deep hypothermia and circulatory arrest (DHCA), remain an issue of debate. This is related in part to the gap between the experimental understanding of brain injury and the clinical application of various neuroprotective strategies. As suggested by Dr Augoustides, this gap can be attributed to the lack of clinically relevant guidelines for effective laboratory study of circulatory arrest.

Our goal was to assess one possible mechanism of neuronal injury after DHCA. Because apoptosis seems to involve a . . . [Full Text of this Article]


Related Article

The conduct of experimental circulatory arrest: The search for clinical relevance
John G.T. Augoustides
J. Thorac. Cardiovasc. Surg. 2007 134: 1381-1382. [Extract] [Full Text] [PDF]





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