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J Thorac Cardiovasc Surg 2009;137:1275-1276
© 2009 The American Association for Thoracic Surgery
Brief Communication |
a Division of Cardiac Surgery, McGill University Health Center, Montreal, Quebec, Canada
b Department of Pathology, McGill University Health Center, Montreal, Quebec, Canada
Received for publication March 12, 2008; revisions received March 12, 2008; accepted for publication March 21, 2008. * Address for reprints: Dominique Shum-Tim, MD, MSc, Division of Cardiothoracic Surgery, McGill University Health Center, 1650 Cedar Avenue, Montreal, Quebec, H3G 1A4, Canada. (Email: dshumtim@yahoo.ca).
| The first 20% of the full text of this article appears below. |
Surgical treatment of active infective valvular endocarditis is associated with substantial morbidity and mortality, particularly when the infection spreads below the primary infected valve. We report the case of a patient in whom acute mitral regurgitation (MR) developed secondary to the rupture of both papillary muscles (PMs) after aortic valve replacement (AVR) for infective endocarditis.
Clinical Summary
A previously healthy 23-year-old morbidly obese man was transferred to our hospital with a 5-day history of left-sided chest pain, dyspnea, cough, and fever. On arrival, his blood pressure was 120/48 mm Hg, with a regular pulse rate of 148 beats/min, a temperature of 38.6°C, a respiratory rate of 26 breaths/min, and an oxygen saturation of 86% on room air. The only remarkable physical findings were a grade III/VI long, diastolic, blowing murmur at the right second interspace and a progressively decreasing level of consciousness, with no focal neurologic signs. After intubation, a transesophageal echocardiogram revealed the presence of a 1 x 3-cm vegetation involving the right and left aortic
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