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J Thorac Cardiovasc Surg 2009;138:1027-1028
© 2009 The American Association for Thoracic Surgery

Brief Technique Report

Totally percutaneous valve replacement for severe aortic regurgitation in a degenerating bioprosthesis

^a Interventional Cardiology, Department of Cardiac Thoracic and Vascular Sciences, University of Padova, Italy
^b Institute of Anaesthesiology, Department of Cardiac Thoracic and Vascular Sciences, University of Padova, Italy
^c Institute of Cardiac Surgery, Department of Cardiac Thoracic and Vascular Sciences, University of Padova, Italy

Received for publication June 9, 2008; accepted for publication July 6, 2008.

^_* Address for reprints: Massimo Napodano, MD, Interventional Cardiology, Department of Cardiac Thoracic and Vascular Sciences, University of Padova, 2 via Giustiniani. 35100. Padova. Italy. (Email: massimo.napodano@gmail.com).

The first 20% of the full text of this article appears below.

	Introduction

 
Recently, the feasibility and safety of percutaneous aortic valve replacement (PAVR) has been reported in the treatment of degenerative aortic valve stenosis in patients at high-risk for surgical aortic valve replacement (AVR).¹ However, so far this therapy has been limited to patients with severe stenosis of the native valve. We report the case of a patient with severe aortic regurgitation owing to bioprosthesis dysfunction who was successfully treated by implantation of a CoreValve (CoreValve Inc, Irvine. Calif) prosthesis with a totally percutaneous approach.

	Clinical Summary

 
An 84-year-old woman, with previous surgical AVR with a bioprosthesis (Biocor 25 mm stentless; (Biocor Industria e Pesguisa Ltda, Belo Horizonte, Brazil) and cardiac pacemaker implantation for severe aortic stenosis in 1998, was admitted to a community hospital because of pulmonary edema. Comorbidity included hypertension, chronic renal failure, and previous left hemicolectomy for bowel malignancy. Transthoracic echocardiogram revealed a severe transprosthetic aortic regurgitation caused by leaflet degeneration and prolapse; the left ventricle was . . . [Full Text of this Article]

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