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J Thorac Cardiovasc Surg 2002;123:615-616
© 2002 The American Association for Thoracic Surgery
Editorials |
From the Center for Pediatric and Congenital Heart Disease, Cleveland Clinic Foundation, Cleveland, Ohio.
Received for publication Oct 4, 2001. Accepted for publication Oct 12, 2001. Address for reprints: Roger B. B. Mee, MB, ChB, FRACS, Center for Pediatric and Congenital Heart Disease, Cleveland Clinic Foundation, 9500 Euclid Ave, M/41, Cleveland, OH 44195 (E-mail: meer@ccf.org).
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Junctional ectopic tachycardia (JET) may severely compromise cardiac output long enough to cause death or multiorgan damage. The hemodynamic impact of JET is probably most severe in those patients who exhibit the most evidence of right, left, or biventricular failure before JET occurs and in those in whom the JET rate most exceeds the heart rate normal for that age group. The heart failure before JET is due to a mosaic of factors: preexisting or new structural lesions inducing volume or pressure loading and preexisting or new myocardial insufficiency (systolic and diastolic dysfunction), with potential exacerbation by high core temperature, inadequate intravascular volume, electrolyte imbalance, and various other factors.
The management of JET is now fairly well
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J. Thorac. Cardiovasc. Surg. 2002 123: 624-630.
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