JTCS
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Videos
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to Personal Folders
Right arrow Download to citation manager
Right arrow Author home page(s):
Filip Rega
Bart Meyns
Right arrow Permission Requests
Google Scholar
Right arrow Articles by Verbelen, T.
Right arrow Articles by Meyns, B.
PubMed
Right arrow PubMed Citation
Right arrow Articles by Verbelen, T.
Right arrow Articles by Meyns, B.
Related Collections
Right arrow Lung - transplantation
Right arrow Cardiac - physiology
Right arrow Congenital - cyanotic

J Thorac Cardiovasc Surg 2013;145:e7-e9
© 2013 The American Association for Thoracic Surgery

Case Report

Acute left ventricular failure after bilateral lung transplantation for idiopathic pulmonary arterial hypertension

Tom Verbelen, MDa,*, Sophie Van Cromphaut, MD, PhDb, Filip Rega, MD, PhDa, Bart Meyns, MD, PhDa

a Department of Cardiac Surgery, University Hospitals Leuven, Leuven, Belgium
b Department of Intensive Care, University Hospitals Leuven, Leuven, Belgium

Received for publication July 6, 2012; revisions received September 12, 2012; accepted for publication October 11, 2012.

* Address for reprints: Tom Verbelen, MD, Department of Cardiac Surgery, University Hospitals Leuven, Herestraat 49, 3000 Leuven, Belgium. (Email: tom.verbelen@med.kuleuven.be).

The first 20% of the full text of this article appears below.


   Introduction
 

Formula Video clip is available online.

 
We report a case of unexpected severe left ventricular (LV) dysfunction in a patient undergoing a bilateral lung transplant (BLTx). We attribute his complicated postoperative course to diastolic dysfunction in a chronically deprived LV in combination with an increased preload, caused by normalization of the pulmonary vascular resistance (PVR), and an increased afterload during weaning.


   Clinical Summary
 
After optimal medical treatment for 12 years, a 23-year-old man with idiopathic pulmonary arterial hypertension had deterioration to New York Heart Association functional class III. Catheterization indicated elevated pulmonary arterial pressure (PAP) values of 121 mm Hg systolic, 72 mm Hg diastolic, and 89 mm Hg mean; a severely reduced cardiac index of 1.63 L/(min · m2); and a PVR of 2309 dynes · s · cm–5. In January 2012, the patient underwent an urgent BLTx because of uncontrollable right ventricular failure (Figure 1 , A). Surgery was uneventful. After the operation, PAP decreased to 30 mm systolic, 20 mm diastolic, and 22 mm Hg mean. PVR decreased to . . . [Full Text of this Article]






HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
ANN THORAC SURG ASIAN CARDIOVASC THORAC ANN EUR J CARDIOTHORAC SURG
J THORAC CARDIOVASC SURG ICVTS ALL CTSNet JOURNALS
Copyright © 2013 by The American Association for Thoracic Surgery.